Need for treatment in all skeletal class III cases – a dilemma? a case report

  • Authors

    • Monika Mahajan Himachal Pradesh government Dental College & HospitalShimla
    2017-10-02
    https://doi.org/10.14419/ijdr.v5i2.8199
  • Class III malocclusion, Genetics, Multifactorial etiology, Skeletal malocclusion, Treatment need.
  • Skeletal malocclusion affects dental and facial tissues. A complicating factor for diagnosis and treatment of skeletal class III malocclusion is its multifactorial etiology. Genetics play an important role in determining the facial morphology of an individual. Prediction of a skeletal class III based on morphology can play an important step in orthodontic diagnosis and treatment planning. This case report further supports the significant role of genetics in skeletal class III malocclusion. As seen in our case the skeletal class III if left untreated does not detiorate but rather shows a decrease in ANB ie in anterioposterior discrepancy. Hence the need for treatment should be analysed thoroughly in skeletal class III patients.

  • References

    1. [1] Profitt WR, Fields HW. Contemporary orthodontics, 4th edition, St.Louis. The CV. Mosby Co; 2000.

      [2] Iwagaki H: Hereditary influence of malocclusion. Am J Orthodontics. 1938; 24:328-36. https://doi.org/10.1016/S0096-6347(38)90140-1.

      [3] Ishii H, Morita S, Takeuchi T, Nakamura S: Treatment effect of combined maxillary protraction and chincup appliance in severe class III cases. Am J Orthod Dentofacial Orthop. 1987; 92: 304-12. https://doi.org/10.1016/0889-5406(87)90331-3.

      [4] Allwright WC, Bundred WH: A survey of handicapping dentofacial anomalies among Chinese in Hong Kong. Internat DJ.1964; 14: 5 05-19.

      [5] Litton SF, Ackermann LV, IssacsonRJ,Shapiro BL: Agenetic study of class 3 malocclusion. J Ortod.1970; 58: 565-77. https://doi.org/10.1016/0002-9416(70)90145-4.

      [6] Harris JE, Kowalski CJ, WatnickSS:Genetic factors in the hape of craniofacial complex. Angle Orthod.1973;43:107-11

      [7] Jacobson A, Evans WG,Preston CB, Sadowsky PL: Mandibular prognathism .Am J Orthod .1974;66:140-71. https://doi.org/10.1016/0002-9416(74)90233-4.

      [8] Haitao Li, Ahmed Mosoud, Lawrence R, Voss. Hybrid Hyrax / quad- helix appliance in the phase I treatment of pseudo- class III malocclusion. Journal of the World Federation of Orthodontists. 2013; 2 (2): e107. https://doi.org/10.1016/j.ejwf.2013.02.003.

      [9] Dietrich UC: Morphological variability of skeletal class 3 relationships as revealed by cephalometric analysis. Rep CongrEurOrthod Soc.1970; 131-43.

      [10] Guyer EC, Ellis EE 3rd, McNamara JA Jr., Behrents RG: components of class III malocclusion in juveniles and adolescents. Angle Orthod.1986; 56: 7-30.

      [11] Ellis E 3rd, McNamara JA. Components of adult class III malocclusion.J Oral Maxillofac Surg.1984; 42: 295-305. https://doi.org/10.1016/0278-2391(84)90109-5.

      [12] Emrich RE, Brodie AG, Blayney JR. prevalence of Class 1,Class 2, and Class 3 malocclusions (Angle) in an urban population. An epidemiological study. J Dent Res. 1965; 44: 947-53. https://doi.org/10.1177/00220345650440053301.

      [13] Jacobson A, Evans WG, Preston CB, Sadowsky PL: Mandibular prognathism. Am J Orthod 1974; 66:140-71? https://doi.org/10.1016/0002-9416(74)90233-4.

      [14] McGuigan DG: The Hapsburgs. London, WH Allen, 1966.

      [15] Harris EF,Johnson MG. Heritability of craniometrics and occlusal variables: a longitudinal sib analysis. Am J Orthod Dentofacial Orthop.1991; 99 (3):258-268. https://doi.org/10.1016/0889-5406(91)70007-J.

      [16] Strohmayer W. Die Vereburg des Hapsburg Familientypus. Nova Acta Leopoldina.1937; 5: 219-296.

      [17] Wolff G, Wienker TF, Sander H. On the genetics of mandibular prognathism: analysis of large European noble families. J Med Genet.1993; 30: 112-116. https://doi.org/10.1136/jmg.30.2.112.

      [18] Suzuki S. Studies on the so called reverse occlusion. J Nihon UnivSch Dent.1961; 3:51-58.

      [19] Horowitz S. Osborne R. De George F. A cephalometric study of craniofacial variation in adults twins. Angle Orthod.1960; 30:1-5.

      [20] Fernex E, Hauenstein P, Roche M.Heredity and craniofacial morphology.Transactions of the European Orthodontic society.1967:239-257.x

      [21] El-Gheriani AA, Maher BS, El- Gheriani AS, ScioteJJ.Segregation analysis of mandibular prognathism in Libua. J Dent Res.2003 Jul; 82 (7): 523-7. https://doi.org/10.1177/154405910308200707.

      [22] Cruz et al. Major gene and multifactorial inheritance of mandibular prognathism. Am J Med Genet A. 2008 Jan 1; 146 a (1):71-7.

      [23] Rakosi T, Schilli W. ClassIII anomalies: a coordinated approach to skeletal, dental, and soft tissues problems. J Oral Surg.1981; 39:860-70.

      [24] MitaniH,SatoK,Sugawara J: Growth of mandibular prognathism after pubertal growth peak. Am J Orthod Dentofacial Orthop.1993; 104: 330-36. https://doi.org/10.1016/S0889-5406(05)81329-0.

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    Mahajan, M. (2017). Need for treatment in all skeletal class III cases – a dilemma? a case report. International Journal of Dental Research, 5(2), 177-181. https://doi.org/10.14419/ijdr.v5i2.8199